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doi: 10.1016/j.bbrc.2015.05.042 240 Schulze-OsthoffKFerrariDRiehemannKWesselborgS

Mechanism involves NO system modulation (homeostatic regulationupregulating when deficient, moderating when excessive), VEGF/VEGFR2 upregulation for angiogenesis, FAK-paxillin pathway activation for cell migration (40% increase per Chang et al.), and EGR-1 transcription factor upregulation for growth factor sensitivity