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Description

PLOS ONE 6 , e22652 (2011)

PINK1-induced mitophagy promotes neuroprotection in Huntingtons disease

The mechanism may be that Lip-1 inhibits the secretion of pro-fibrotic factors by renal tubular epithelial cells, ultimately suppressing the activation of fibroblasts surrounding the renal tubules

Meanwhile, in RAAS, ACE's conversion of angiotensin I to angiotensin II, which has a potent vasoconstrictor impact, may cause the adrenal cortex to generate more aldosterone, both of which raise blood pressure