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For example, Liu and collaborators observed similar effects to those described in HCC, but in pancreatic cancer cells (Liu et al., 2018 [19])

These are the same pathways cancer cells exploit to grow and spread

This would support either camp of thought around whether PD pathology is driven by a proteinopathy (toxic accrual of S aggregates gain of function) or a proteinopenia (loss of S native function)