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Intravenous (IV) Therapy 5.4.1.3.6

Using the molecular classification of glioblastoma to inform personalized treatment

A recent work has shown that epileptic seizures lead to overexpression of Keap1 promoting proteasomal degradation of NRF-2 in neurons, while inhibition of Keap1 expression stabilizes NRF-2, and increases the level of GSH, GPX4, superoxide dismutase (SOD), and the cystine/glutamate exchanger subunit SLC7A11 [47]