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Its potential influence on these crucial processes renders it a potentially powerful tool in better understanding cellular aging and in developing novel treatments in regenerative medicine and age-related health problems

This provides supportive, if circumstantial, evidence for a role for UA in protecting against the neuronal degeneration underlying PD

This is a preview of subscription content, access via your institution Access options Access Nature and 54 other Nature Portfolio journals Get Nature+, our best-value online-access subscription $32.99 / 30 days cancel any time Subscribe to this journal Receive 12 digital issues and online access to articles $119.00 per year only $9.92 per issue Rent or buy this article Prices vary by article type from$1.95 to$39.95 Prices may be subject to local taxes which are calculated during checkout References Bloomfield, G

The Arg3 to Leu3 substitution disrupts a key contact residue in the IGFBP binding interface, and the N-terminal extension further sterically interferes with binding pocket access