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In support of this, AMPK modulates the protein expression of acetyl CoA carboxylase (ACC), which facilitates the transport of long-chain fatty acids into the mitochondria for -oxidation (49), thus supporting impaired fatty acid oxidation in taurine-deficient conditions

Iron overload may not be a central driver of ferroptosis in these disorders, which challenges current assumptions about the physiological triggers of ferroptosis in vivo [35]

However, intense overtraining can actually deplete your stores, so balance is key