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Whether via dietary precursors (e.g., N-acetylcysteine), targeted transdermal delivery, or enhancement of cysteine transport, interventions that optimize glutathione metabolism represent promising strategies for clinical brain health support

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The results showed that JA induced the production of NO, enhanced the phosphorylation level of MEK1/2, increased the catalytic activity of the GLDH enzyme and enzymes of the ascorbate-glutathione pathway, then increasing the AsA and glutathione (GSH) contents

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