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Glial cells to a very great extent define neuropathology, its progression and outcome: as long as glial defence prevails, the pathological process is resolved, whereas the failure of glial defence results in neuronal death and neurological deficits

These limitations reduce the clinical applicability of fast-releasing H 2 S-donors [117,118]

Hyperglycemia and IR are responsible for worsening S aggregation and mitochondrial dysfunction, both of which contribute to the loss of dopamine-producing neurons

This summary provides basic information about Tirzepatide but does not include all information known about this medicine