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Saudi Pharmaceutical Journal 31(9):101745 Lee Y, Kim H, Lee E, Hahn H, Heo Y, Jang DM, Kwak K, Kim HJ, Kim HS (2023) Structural insights into N-terminal methionine cleavage by the human mitochondrial methionine aminopeptidase, MetAP1D

These include: Drugs called H2 receptor antagonists Metformin Proton pump inhibitors

Overall, the triple-drug combination was effective in inducing cell death across four high-risk NB cell lines

Initially, the mechanisms of action of this enzyme were considered paradoxical because CD38 is predominantly localized on the cell surface, yet its final products are utilized within cytoplasmic compartments