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Results l-Carnitine attenuated the increased ROS levels in the cytosol and mitochondria of PFOS-treated RTCs and the cell death of such cells We previously demonstrated that N-acetylcysteine (NAC), a ROS scavenger, rescues RTCs from PFOS-mediated apoptosis triggered by autophagy activation because the autophagy inhibition due to 3-methyladenine can block the aforementioned apoptosis 3

Aryl-hydrocarbon receptor-dependent alteration of FAK/RhoA in the inhibition of HUVEC motility by 3-methylcholanthrene

Moreover, the comparison of CRP levels at the different timepoints evaluated did not show any significant differences between the LCLT and placebo groups, as presented in Table 3

The primary role of L-Carnitine is to shuttle fatty acids into the mitochondria for energy production