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The network view showed the most significant relationships between perturbed biochemical pathways in PsA, for example, between glutathione, alanine metabolism, and glycine and serine metabolism via glutamate metabolism, as well as between glucose-alanine cycle and alanine metabolism via the transfer of acetyl groups into mitochondria (Fig

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A hypothesized mechanism is attributed to L-cells that co-produce GLP-1 and GLP-2 at the same time, which regenerates intestinal epithelium, mediates peptide YY production, and subsequently, through appetite suppression, prolongs satiety through GLP-1 receptors and thus manages weight control [33, 47]