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SAHA sensitized the cells by increasing the expression levels of proapoptotic regulators, caspases, TNF/TNFR family and death domain proteins (TNFSF10, TNFRSF10B, 11B), and reduction of antiapoptotic signaling ( in vitro data suggest that HDACi might overcome acquired dual resistance and immunosensitize BRAF V600E mutated cells to CTL and NK-killing

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These opposite results may partly attributed to colonization at birth, age, gender, diet, and other factors (Gilbert et al., 2018)