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and (4) growth hormone receptor upregulation at injury sites, restoring local GHR density without elevating systemic GH levels

In order to validate this dopamine-depletion model, we had animals from both vehicle and AMPT groups placed in open-field chambers on three consecutive days to record their baseline locomotor activity

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Interestingly, TNF- is significantly correlated with both apoptotic markers and angiogenic factors: it downregulates BCL2 and upregulates BAX and acts synergistically with HIF-1 to increase VEGF production under ischemic conditions 18,19