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FH and FHL-1 bind to C3b, a component of C3 convertases, which recruits complement protein factor I to degrade C3b and inhibit the formation of these convertases and inactivates the complement system (3)

These findings align with a meta-analysis comparing first-line phase 3 trials, where pembrolizumab plus lenvatinib demonstrated the highest probability of G3-G4 AEs according to the surface under the cumulative ranking curve (SUCRA) analysis, followed by nivolumab plus cabozantinib

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"Regulation of the cystic fibrosis transmembrane conductance regulator ClK channel by its R domain"