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In the face of controversies and challenges, it is essential to establish a more efficient translational interface between science, technology, and clinical practice, driving mitochondrial metabolic reprogramming therapy from a potential pathway to clinical reality. Conclusion Mitochondrial metabolic reprogramming plays a central role in the therapeutic failure of CRC

In this process by the usage of mixed functional ligands one can precisely tune the physical and chemical properties of those metal complexes

Two to six times more ynfA gene expression than tolC gene expression was observed (Sarkar et al., 2015)