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Deep phenotyping of myalgic encephalomyelitis/chronic fatigue syndrome in Japanese population

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Moreover, the appearance of both HuR cleavage products and higher-molecular weight species in the absence of caspase-3 activation suggests that HuR undergoes caspase-independent modification during APAP-induced hepatotoxicity, consistent with the predominance of necrotic cell death in this model

Through controlling these targets, HSF1 acts in diverse stress-induced cellular processes and molecular mechanisms, including the endoplasmic reticulum unfolded protein response and ubiquitinproteasome system, multidrug resistance, autophagy, apoptosis, immune response, cell growth arrest, differentiation underlying developmental diapause, chromatin remodelling, cancer development, and ageing