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Meanwhile, methyltransferase 3-mediated m6A modification promotes the degradation of synaptosomal-associated protein 29 mRNA, thereby impairing the restoration of autophagic flux, triggering mitochondrial crisis and excessive ROS accumulation, and ultimately exacerbating oxidative stress injury in the ischemic microenvironment (48)
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[Abstract]2024 Jun 16:137:112480
