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H 2 O 2 produced in higher levels from injured microglia increases ROS production which proceeds to cause oxidative stress and aftermath features like DNA fragmentation, protein oxidation, lipid peroxidation, apoptosis

My writing has slowed down during the pandemic since my main impetus derives from reporting on science progress at Stanford, which was closed from March, 2020 to October, 2021

One particularly instructive example comes from research using 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced models of PD, where chronic administration of monoacylglycerol lipase inhibitors was found to not only preserve dopaminergic neurons in the substantia nigra but also induce a distinctive microglial activation profile characterized by increased release of neurotrophic factors and decreased expression of classical inflammatory markers [459]

Identity is confirmed by matching the measured mass to the theoretical molecular weight of 1419.55 g/mol, which also flags any truncation in synthesis