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Regarding the pathogenesis of COVID-19, the role of the Th1 cell subset, which plays a key role in the immune response against intracellular pathogens, is quite controversial

Addressing these gaps, particularly those involving redox-sensitive pathways, mitochondrial integrity, and growth-factormediated protection, will be essential for optimizing NSC-based therapeutic strategies and identifying reliable biomarkers of NSC function in neurodegenerative disease