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Description

N.AmoscatoA

Urology 1997

56 aimed to improve cisplatin-induced kidney damage by reducing oxidative stress and apoptosis with sinapic acid, observing a significant reduction in kidney damage following sinapic acid treatment

Moreover, genetic inactivation of the tumor suppressor NF2, a frequent tumorigenic event in mesothelioma, renders cancer cells more sensitive to ferroptosis, malignant mutations in NF2YAP signaling can be used to predict the responsiveness of cancer cells to future ferroptosis-inducing therapy [56]