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Their findings confirm the results of the meta-analysis (involving more than 47,500 subjects) in which overall lack of association between GSTM1 genotypes and coronary heart diseases was shown

Formyl peptides have been shown to selectively activate phospholipase A2 (PLA 2 ) in neutrophils phosphorylated by p38 MAPK-activated kinase, and PLA 2 inhibitors block neutrophil chemotaxis toward formyl peptides but not toward CXCL8