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Description

Methylcobalamin acts as a powerful neuroprotective agent by upregulating the lipid synthesis of Schwann cells, the specialized glial cells responsible for producing myelin in the peripheral nervous system

Microscopically, there are no more inflammatory infiltrate, well-oriented recovered tissue of musculotendon junction appears in BPC 157 treated rats at the 28 days and 42 days

In addition to dopaminergic neuronal loss in the SN and the onset of motor symptoms in PD, these changes cause microgliosis, mitochondrial failure, oxidative stress, and inflammation