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Journal of Orthopaedic Research, 21(4), 753-758

Jan 10, 2026 Tissue repair mechanisms operate through multiple interconnected pathways that rarely respond optimally to single interventions, with injuries activating inflammatory cascades, angiogenic processes, fibroblast proliferation, and extracellular matrix remodeling simultaneously across damaged regions

This mechanism is relevant to neuropathy because NF-kB-driven inflammation is a central mediator of nerve damage in diabetic neuropathy, autoimmune neuropathies, and chemotherapy-induced peripheral neuropathy