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The degradation of branched-chain amino acids mainly occurs in the mitochondrial matrix, contributing to energy metabolism, mitochondrial biogenesis, as well as protein quality control in both mitochondria and cytosol

This was matched by an increase in genes of the catechol branch of beta-ketoadipate pathway, which may be related to the proteobacterial degradation of B12 as previously shown in Pseudomonas species [56]

The relationship between fatty liver disease and vitamin B12 deficiency is complex

BPC-157 stimulated earlier maturation of granulation tissue and resulted in more organized collagen compared to becaplermin treatment