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ROS-dependent nerve dysfunction seems to be related to neurovascular defects associated with hypoxia.21 Increased activity of NADPH oxidase has been detected in the retina of diabetic rats, thus suggesting a role of increased oxidative stress also in retinopathy.22 In patients with diabetes, endothelial cells are the main source of ROS, which overwhelm the cellular antioxidant capacity and impair cellular function.23 Impaired endothelium dependent vasodilatation has been demonstrated in vascular beds in patients with type 1 and type 2 diabetes.24 In addition, vascular smooth muscle cell function is impaired with a reduction of response to circulating vasodilators in subjects with diabetes mellitus.25 Oxidative stress also induces proliferation of vascular smooth muscle cells and promotes their migration into atherosclerotic lesions

L-glutathione and vitamin C are not only resistant to heat and stomach acid thanks to liposomal technology, but they are also gentle on the stomach and can be effectively absorbed by the intestinal cells due to the lipid coating

Individuals diagnosed with BD have an approximate mortality rate that is approximately twice that of the general population

Phase 3 clinical trials demonstrated a significant reduction in visceral fat without major metabolic side effects (Falutz et al., J Clin Endocrinol Metab 2010)