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[DOI] [PMC free article] [PubMed] [Google Scholar] 130.Sumi N., Nishioku T., Takata F., Matsumoto J., Watanabe T., Shuto H., Yamauchi A., Dohgu S., Kataoka Y

M., Smith S

Conclusion Collectively, we unveil GPR68 as a critical sensor of ischemic acidosis that orchestrates ferroptosis resistance via PI3K/Akt/Nrf2 signaling