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Later on, the molecular genetic basis of the disease was linked to a transition mutation in the snca gene A53T which results in the substitution of alanine to threonine in the N terminal 53rd amino acid of the protein leading to its abnormal aggregation (fibrillation) (Lee and Trojanowski, 2006)

Thomsen JL et al (2015): Premature awakening and underuse of neuromuscular monitoring in a registry of patients with butyrylcholinesterase deficiency

Micronutrients such as vitamins, minerals and trace elements are responsible for many metabolic processes such as cell renewal, metabolism and maintaining organ function