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Further interventional studies conducted on humans are needed to clear the causal relationship between T1D and intestinal microbiota [60]

The latter in response to stimulation by intestinal microbial antigens, particularly LPS, upregulate the production of proinflammatory cytokines (e.g., IL-1, IL-6, TNF) and reactive oxygen species (ROS), contributing to the enhancement of pro-inflammatory reactions (Hine and Loke, 2020)