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[5] This surge in carnitine production enhances the body's ability to transport fatty acids into mitochondria for energy metabolism, potentially supporting fat oxidation during training

Inhibition of PFKFB3 impairs both tip cell selection and stalk cell proliferation

A.WoellnerC.SalzerU.et al

The activated JNK [phospho-c-Jun N-terminal kinase (p-JNK)] combines with its target SH3 domain-binding protein (Sab) on the mitochondrial membrane, leading to a self-sustaining activation circuit of ROS production and JNK activation, which eventually leads to the opening of mitochondrial membrane permeability transition pore (MPT) [61]